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Overall, these findings demonstrate that TSP, induced by NRF2 under cystine-limiting conditions, plays a crucial role in promoting quiescent cell tolerance to ferroptosis triggered by cystine deprivation (Fig

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proposed that TGFBR1 overexpression aggravates retinal inflammation via microglial ferroptosis, yet a contradictory finding indicates no significant association between ferroptosis-related genes and disease progression, suggesting that immune cell infiltration occurs independently of ferroptosis (45, 156)