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Focal segmental glomerulosclerosis (FSGS) paradigms demonstrate enhanced Bifidobacterium , Collinsella , and Candida colonization, while experiencing significant depletion of beneficial taxa including Granulicatella, Christensenella, Turicibacter , and Rikenella , culminating in elevated deleterious metabolite concentrations, compromised intestinal barrier integrity, endotoxemia, and sustained mTORC1 signaling pathway activation that perpetuates glomerular sclerosis and fibrogenesis (Parker et al., 2020
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