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These findings suggest that pathological cardiac pressure overload induces NNT reverse functional mode to generate NADH at the expense of NADPH resulting in a high NADH/NAD + and reductive stress, further promoting ROS at complex I leading to oxidative injury and cell death (Fig
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l-glutathione (N(N-l-γ-glutamyl-L-cysteinyl) glycine, GSH) is a tripeptide composed of cysteine, glutamic acid, and glycine ( In this study, we used GSH rinse as new technology to modify the GLUT-fixed bovine pericardium (BP), thereby addressing the issues of toxicity and calcification
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