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In an LPS-induced depression model, AAV-GFAP-Sig-1R-induced astrocyte-specific overexpression of Sig-1R significantly reduces the expression of the astrocyte activation marker GFAP, increases BDNF, and attenuates the LPS-induced increase in mRNA levels of inflammatory mediators such as TNF-α, IL-1β, and iNOS (Guo et al., 2021)