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The formation of mitochondrial ROS upon reperfusion is considered an early event, presumably initiated by succinate accumulation during ischemia, which is rapidly re-oxidized upon reperfusion by succinate dehydrogenase, thereby generating a burst of superoxide by reverse electron transport at mitochondrial complex I [499]

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It is further reported that APX gene expression is affected by abiotic stresses like salinity, drought, light fluctuations and abcisic acid (ABA) production (Bonifacio et al., 2011