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This study also highlighted a promise of NLCs in the treatment application of AD via an intranasal route, as evidenced by increased accumulation and prolonged retention time of quercetin-loaded NLCs in the nasal cavity (~8 h) (Satpathy et al., 2025)
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Moreover, in TNF-α-stimulated ECs, the copper-dependent ATOX1-TRAF4 interaction promotes nuclear translocation and initiates ROS-dependent inflammatory responses, solidifying this axis as a promising therapeutic target [45]
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