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120,121 Notably, estrogen induces expansion of breast cancer stem-like cell subpopulations and can lead to tumorigenic phenotypes in vivo through a paracrine FGF–FGFR–Tbx3 signaling pathway, which happens also to modulate normal embryonic breast stem cells, whereas cotreatment of cells with tamoxifen or a small molecule inhibitor of fibroblast growth factor receptor (FGFR) signaling was capable of preventing estrogen-induced expansion of cancer stem cells

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