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Moreover, genetic polymorphisms in metabolic enzymes, such as cytochrome P4501A2 (CYP1A2) [42], adenosine receptors [43], and transcription factor Nrf2 [44], can account for individual variations in caffeine metabolism and response

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At low, physiologically buffered concentrations, they mediate oxidative eustress, a form of beneficial stress that drives adaptive responses in cell survival, metabolic plasticity, and immune homeostasis