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These findings are consistent with the potential role of glutathione reductase in regulating apoptotic cell death by altering concentrations of glutathione in mitochondria

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However, a growing body of evidence reveals a pronounced immunoinflammatory phenotype in OA, wherein macrophages, T/B lymphocytes, and inflammatory factors such as IL-1β, TNF-α, and IL-6 play a critical role in the local microenvironment (105)