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Mucin 5AC serves as the nexus for β-catenin/c-myc interplay to promote glutamine dependency during pancreatic cancer chemoresistance

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These beneficial effects may be due to the formation of detour circuits—which encompass spared tissue surrounding the lesion—and could reconnect locomotor circuits [69], thus enabling afferent inputs to be processed and conveyed to the cortex [73] and improving spinal reflexes, even below the injury [74]